Researchers from Osaka University found that lifestyle diseases such as diabetes, high cholesterol, and fatty liver are caused by age-related changes in fat cells. Loss of a protein called Rubicon in aged adipocytes allows intracellular degradation processes to kick into overdrive, disrupting normal cellular function and causing the onset of lifestyle diseases. By inhibiting this degradation and restoring a more youthful cellular environment, we may be able to prevent the onset of lifestyle diseases.
- Trying to beat a coke habit with cannabis? Not so fast !
- Secrets of traumatic stress hidden in the brain are exposed
- Pace of prehistoric human innovation could be revealed by ‘linguistic thermometer’
- Harpy eagles could be under greater threat than previously thought
- Children can bypass age verification procedures in popular social media apps